Prof. Dr. Balázs Csaba
Endokrinológus
A Budai Endokrinközpont specialistája "endokrinológia" témakörben
Kérem, tegye fel kérdéseit, készséggel válaszolok Önnek! Bizalmát köszönöm!
Endokrinológus
A Budai Endokrinközpont specialistája "endokrinológia" témakörben
Kérem, tegye fel kérdéseit, készséggel válaszolok Önnek! Bizalmát köszönöm!
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Kérdezz-felelek
Kérdezni a gomb megnyomásával tudsz, amennyiben a napi kérdések száma még nem haladta meg a napi limitet.
Dear Dr. Balazs,
I am writing to ask your advice regarding treatment for hyperthyroidism. My mother (age 78) has been diagnozed with hyperthyroidism which she developed after my father's death in March 2011. She has been presented with the option of surgery or radioactive iodine treatment by her doctor after a series of tests. At the moment she is a patient at the St. Janos Hospital in Budapest. I feel she should be getting a second opinion and wondered whether you can suggest how we can find a specialist in this field in Budapest to help her make an informed decision.
Any advice you can give is much appreciated.
Kind regards,
H. Elias
I am writing to ask your advice regarding treatment for hyperthyroidism. My mother (age 78) has been diagnozed with hyperthyroidism which she developed after my father's death in March 2011. She has been presented with the option of surgery or radioactive iodine treatment by her doctor after a series of tests. At the moment she is a patient at the St. Janos Hospital in Budapest. I feel she should be getting a second opinion and wondered whether you can suggest how we can find a specialist in this field in Budapest to help her make an informed decision.
Any advice you can give is much appreciated.
Kind regards,
H. Elias
Dear H. Elias,
The therapy of hyperthyroid syndrome depends on the etiology of hyperfunction. Taking into account her age, the radioiodine therapy seems to be advisable.
I enclosed the recent published guideline of hyperthyroidism as well.
If the hyperthyroidism of autoimmune origine, the other management is concidered.
Because recently an increasing amount of data has been gathered on the connection between neuro-endocrine and immune systems. Results of molecular genetic research provided evidence for a common language of these systems including neurotransmitters, hormones and cytokines. It has been proven that the immune system is capable of producing neurotransmitters and hormones and even the endocrine system can prepare cytokines. This integrative (holistic) approach makes possible the investigation of physiological and pathological events as interactions of psycho-neuro-endocrine-immune systems. The associations of autoimmune diseases and the autoimmune polyendocrine syndromes constitute a heterogeneous group of disorders characterised by decreased or lost immune tolerance against self-antigens. Molecular genetic research explored the mechanism of the associations of diseases which are called organ-specific. Autoimmune polyendocrine syndrome Type 1 is characterised by the presence of at least two of the three cardinal diseases: Addison’s disease, autoimmune hypoparathyroidism, and mucocutaneous candidiasis. This rare autosomal recessive syndrome is induced by mutations of the autoimmune regulator (AIRE) gene. Autoimmune polyendocrine syndrome Type 2 that occurs at a much higher frequency is observed and defined as the coexistence of Addison’s disease, autoimmune thyroid disease and/or Type 1 diabetes mellitus. Autoimmune polyendocrine syndrome Type 3 is characterised by an association of autoimmune thyroid disease and Type 1 diabetes mellitus. In contrast to autoimmune polyendocrine syndrome Type 1, HLA and other antigens have proven to be important in Types 2 and 3 of the syndrome. Identification of genetic factors predisposing to these syndromes contributes to our understanding of the common mechanisms involved in autoimmunity and offers a possibility for early treatment and prevention as well.
With best regards:
Prof. Csaba Balázs M.D. D.Sc.
Prof. Dr. Balázs Csaba
2011-10-26 12:56:51
The therapy of hyperthyroid syndrome depends on the etiology of hyperfunction. Taking into account her age, the radioiodine therapy seems to be advisable.
I enclosed the recent published guideline of hyperthyroidism as well.
If the hyperthyroidism of autoimmune origine, the other management is concidered.
Because recently an increasing amount of data has been gathered on the connection between neuro-endocrine and immune systems. Results of molecular genetic research provided evidence for a common language of these systems including neurotransmitters, hormones and cytokines. It has been proven that the immune system is capable of producing neurotransmitters and hormones and even the endocrine system can prepare cytokines. This integrative (holistic) approach makes possible the investigation of physiological and pathological events as interactions of psycho-neuro-endocrine-immune systems. The associations of autoimmune diseases and the autoimmune polyendocrine syndromes constitute a heterogeneous group of disorders characterised by decreased or lost immune tolerance against self-antigens. Molecular genetic research explored the mechanism of the associations of diseases which are called organ-specific. Autoimmune polyendocrine syndrome Type 1 is characterised by the presence of at least two of the three cardinal diseases: Addison’s disease, autoimmune hypoparathyroidism, and mucocutaneous candidiasis. This rare autosomal recessive syndrome is induced by mutations of the autoimmune regulator (AIRE) gene. Autoimmune polyendocrine syndrome Type 2 that occurs at a much higher frequency is observed and defined as the coexistence of Addison’s disease, autoimmune thyroid disease and/or Type 1 diabetes mellitus. Autoimmune polyendocrine syndrome Type 3 is characterised by an association of autoimmune thyroid disease and Type 1 diabetes mellitus. In contrast to autoimmune polyendocrine syndrome Type 1, HLA and other antigens have proven to be important in Types 2 and 3 of the syndrome. Identification of genetic factors predisposing to these syndromes contributes to our understanding of the common mechanisms involved in autoimmunity and offers a possibility for early treatment and prevention as well.
With best regards:
Prof. Csaba Balázs M.D. D.Sc.
Prof. Dr. Balázs Csaba
2011-10-26 12:56:51
Olvasói értékelés: nincs még értékelés
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